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Impact of prenatal risk factors in attention deficit hyperactivity disorders: Potential for gene-environment interactions
Uppsala universitet, Institutionen för psykologi.ORCID-id: 0000-0003-1209-8802
2008 (Engelska)Ingår i: Psychiatry, ISSN 0033-2747, E-ISSN 1943-281X, Vol. 7, nr 12, s. 516-519Artikel i tidskrift (Refereegranskat) Published
Abstract [en]

Attention deficit hyperactivity disorder (ADHD) is a multifactorial disorder and both genetic and environmental factors have been implicated in its etiology. Yet, the interaction between genes and environment is seldom studied directly. This article considers the plausibility of nicotine exposure during prenatal development as well as postnatal factors in the etiology of ADHD. The few existent studies show inconsistent results, but provide preliminary evidence suggesting that nicotine exposure together with genes in the dopaminergic system confer risk for ADHD. Factors affecting resilience during prenatal and postnatal development remain virtually unexplored. Recommendations for future research are provided.

Ort, förlag, år, upplaga, sidor
Elsevier , 2008. Vol. 7, nr 12, s. 516-519
Nyckelord [en]
ADHD, environmental, etiology, gene-environment interaction, genetic, maternal smoking, methodological issues, postnatal, prenatal, resilience
Nationell ämneskategori
Psykologi
Identifikatorer
URN: urn:nbn:se:miun:diva-16817DOI: 10.1016/j.mppsy.2008.10.004OAI: oai:DiVA.org:miun-16817DiVA, id: diva2:556394
Tillgänglig från: 2012-09-25 Skapad: 2012-08-24 Senast uppdaterad: 2017-12-07Bibliografiskt granskad

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